GoKawiilStatistics and reproducibility
Supplementary Table 5 lists the number of independent samples, brains or hemispheres that we examined. All samples are biological replicates. Results were consistent across samples. In cases in which lineages were labelled, numbers indicate total samples collected; however, in some cases a given lineage might not be labelled in every brain. Data grouped together in the same row are from the same samples. If a figure panel is not labelled here, the data are present in its legend.
Number of samples and statistics for Extended Data Fig. 9
For Extended Data Fig. 9e, n = 17 control, 11 mutant and 4 non-induced hemispheres (TfAP-2 RNAi versus control P < 0.0001; TfAP-2 RNAi versus not induced P = 0.0004; control versus not induced P = 0.9989). For Extended Data Fig. 9e′, n = 17 control, 11 mutant and 5 non-induced hemispheres (TfAP-2 RNAi versus control, P < 0.0001; TfAP-2 RNAi versus not induced, P < 0.0001; control versus not induced, P = 0.3209. For Extended Data Fig. 9e,e′: significance, one-way ANOVA with Tukey’s multiple comparisons test.
For Extended Data Fig. 9f,f′,k,k′,l,l′,q,q′,r,r′,s,s′,t,t′,x,y,y′, significance two-sided, unpaired t-test.
For Extended Data Fig. 9f, n = 17 control, 13 mutant and 1 non-induced hemispheres (TfAP-2 RNAi versus control P = 0.0016).
For Extended Data Fig. 9f′, n = 19 control, 14 mutant and 1 non-induced hemispheres (TfAP-2 RNAi versus control P < 0.0001).
For Extended Data Fig. 9k, n = 10 control and 10 experimental hemispheres (P = 0.0043).
For Extended Data Fig. 9k′, n = 6 control and 8 experimental hemispheres (P = 0.0444).
For Extended Data Fig. 9l, n = 16 control and 12 experimental hemispheres (P < 0.0001).
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